Radiculitis in show jumping horses is the inflammation or irritation of a spinal nerve root, which causes pain, weakness and neuromuscular alterations that can affect the horse's performance. In this discipline, where the spine supports a great deal of impact and flexion, radiculitis can be an important cause of resistance to work, lack of impulsion or pain in the dorsal or lumbar region.
Repetitive impact and mechanical overload: Constant repetition of jumps and landings can generate microtraumas in the spine, affecting the nerve roots.
Structural vertebral problems: Conditions such as kissing spine syndrome, spondylosis, facet arthritis or vertebral subluxations can compress nerve roots.
Neurogenic inflammation: Release of proinflammatory cytokines that sensitize root nociceptors.
Muscular and postural imbalances: Poor biomechanics or weak core musculature can predispose to spinal inflammation.
Trauma or falls: An improper landing or a blow can generate inflammation and compression of the nerve roots.
Biomechanical alterations: Functional overload due to core weakness, pelvic asymmetry or musculoskeletal compensations.
Extrinsic factors: Poorly adjusted saddles, inadequate training techniques and unstable ground.
Symptoms in jumping horses
Radiculitis manifests with clinical signs that depend on the spinal segment affected: Thoracic and upper lumbar region (T10-L2):
Pain on palpation of the spinous processes.
Resistance to dorsal and lateral flexion.
Behavioral changes, such as resistance to work, refusal to jump or irritability.
Decreased hindfoot engagement and difficulty in swinging. Lower lumbar and sacral region (L3-S1):
Loss of power in impulsion.
Incoordination in jumping mechanics.
Sensitivity in the sacroiliac joint and base of the tail.
Clinical and therapeutic management
Treatment should be multimodal, addressing inflammation, pain and underlying biomechanics:
Pharmacologic management
Anti-inflammatory drugs: NSAIDs (flunixin, meloxicam) for acute pain control.
Epidural corticosteroids: methylprednisolone in ultrasound-guided infiltrations.
Neuromodulators: neuropathic hyperalgesia.
Regenerative therapies: Platelet-rich plasma (PRP) or mesenchymal stem cells in chronic injuries.
Biomechanical management and rehabilitation Specific physiotherapy:
Electrostimulation and neuromuscular exercises to strengthen the core.
Passive and dynamic stretching to improve spinal flexibility.